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GSD Information Links and Articles 

DDR titles article 

The Love for these beautiful dogs run deep! This is my grandmother and her dog!


Understanding the Rating System of the DDR GSD's 

Spay and Neutering article

DDR/German Shepherd Information

What you need to know before owning a GSD

The Love for these beautiful dogs run deep! This is my grandmother and her dog!


Author on feeding raw 

How Much Does A German Shepherd Cost Annually?

The cost of dog-keeping varies considerably depending on your German Shepherd (including size, age and overall health), the quality of supplies and equipment,and even where you live. The following numbers should give you an idea of what to expect.

For A German Shepherd Puppy In Her First Year:

  • Veterinary care, including general care and laboratory tests: $100 – $200

  • Immunizations: $50 – $100

  • Spay or neuter: $50 – $200

  • Internal and external parasite treatment and control: $100 – $150

  • Food: $150 – $250

  • Miscellaneous expenses, including collar, leash, bowls, toys, grooming supplies and obedience training: $200 – $225

Total: $650 to $1,125 for the first year only. Note that this total does not include the cost of the puppy.

For An Adult Dog The Annual Costs Would Involve:

  • Veterinary care, including general care and laboratory tests: $50 – $125

  • Immunizations: $40 – $75

  • Internal and external parasite treatment and control: $100 – $150

  • Food: $200 – $400

  • Miscellaneous expenses: $100 – $125

Total: $490 to $875 per year.

These figures are estimates only and they do not include expenses related to illnesses, injuries, showing, competition, breeding, boarding or travel. Senior dogs usually require more health care than adults or puppies.From these figures, it’s clear that the care of a reasonably healthy German Shepherd living to the age of 12 to 14 years could easily cost $7,000 to $15,000 or more.

Article on what Socialization is and isn't

What socialization is and isn't article         


The good dog training and rehabilitation (Facebook)

Okay, so this is a word that gets an awful lot of play in the training world, and it’s definition has come to mean an awful lot of things. Many of which, are incredibly counter-productive, if not downright dangerous.

So let’s see if we can’t clarify a few things.

Socialization isn’t:
-About letting your dog freely interact with dogs at the dog park, day care, or with friends dogs.
-About allowing your dog to meet other dogs on-leash.
-About allowing all manner of people, in all manner of mental/emotional states interact/pet/pressure your dog.
-About exposing your dog to the sights and sounds of cars, buses, motorcycles, bikes, skateboards, joggers...and allowing them to freak out, panic, aggress, hide, bark etc.
-About exposing your dog to the sights and sounds of dogs, cats, and other animals, and allowing them to freak out, aggress, lunge, bark, growl etc.

Socialization is:
-About teaching your dog the proper responses to dogs. What is and isn’t appropriate behavior, and correcting the unwanted when it appears.
-About teaching your dog to walk by the barking, lunging dog(s) on walks and ignore them, completely. Correcting if necessary to achieve this result.
-About advocating for your dog and ensuring people aren’t allowed to pressure your dog, by touching, crouching down, attempting “kisses” etc. That means being a big boy or girl, and stopping others from engaging in unwanted, uninvited interactions.
-About exposing your dog to all manner of daily life “things” and ensuring a proper response. If aggression/arousal is present, it’s corrected, if fear/arousal is present (and causes an overreaction/fleeing etc.) it’s corrected. Ask your dog to learn to ignore and not care about these “life” distractions/concerns/temptations. Teach them to listen to the training, not the world around them.
-About teaching your dog to leave other creatures alone. The cat, the bird, the cow, the goat, the other dog, is simply none of their business. If they decide those things are their business, it’s your job to correct and clarify what is and isn’t their business for them.

Socialization has become a ridiculously simplified, dumbed down, all-encompassing idea. Free interaction and exposure have been presented as a panacea, the magic gateway to a balanced dog. That’s a whole lot of B.S. you’ve been sold, by a lot of people full of B.S.  🙂

Socialization is all about teaching your dog how to behave and exist in the world...properly. People have a belief that only interactions create a well socialized dog. They don’t understand that existence is almost always preferable, and more valuable than actual interaction. Yes exposure is critical, but exposure without 100% clear guidance, and corrections for poor choices, isn’t socialization, it’s chaos, and it’s not teaching your dog what’s right, what’s wrong, and that you’ll keep them safe, so they don’t have to.

A well socialized dog isn’t fazed by the world around them. And that doesn’t come from simple exposure and interactions without guidance. Ironically, that’s precisely how you create anti-social dogs.

Think on that for a minute.

Hip Dysplasia Study 


The 10 most important things to know about canine hip dysplasia



By Carol Beuchat PhD


Hip dysplasia is a hot topic in dogs, if it's possible to stay "hot" for 50 years. Researchers have been working hard for decades looking for solutions, and breeders have been doing their best to reduce the risk of producing affected puppies. But still the problem remains.


There are some simple things we could do to reduce the incidence of hip dysplasia now if we understand a few basic things. Here are the 10 most important things you need to know.


1) All puppies are born with perfectly normal hips

Hip dysplasia is not a congenital defect; it is not present at birth. Multiple studies have demonstrated that all normal puppies are born with "perfect" hips; that is, they are "normal" for a newborn with no signs of dysplasia. The structures of the hip joint are cartilage at birth and only become bone as the puppy grows. If a puppy is going to develop hip dysplasia, the process begins shortly after birth.

This is the hip joint of a 1 day old puppy. The cartilage tissue does not show up on an x-ray until the minerals are deposited that form bone. Proper development of the joint depends on maintaining the proper fit between the head of the femur and the socket (acetabulum).


"The hip joints of all dogs are normal at birth. The joints continue to develop normally as long as full congruity is maintained between the acetabulum and the femoral head... The acetabular rims are stimulated to grow by mild traction applied by the joint capsule and gluteal muscles attached along their dorsal borders, and from pressure by the femoral heads upon the articular surfaces... The morphologic characteristics of the complex hip structure show that biomechanical behavior is the prime influence in the growth of this joint." (Riser 1985)


2) The genes that cause hip dysplasia remain a mystery

Hip dysplasia tends to be more common in some breeds than others and in some lines than others, which indicates that there is a genetic component to the disorder. However, scientists have been looking for genes that are responsible for the development of hip dysplasia in dogs for decades without success.


​Genes that are associated with hip dysplasia have been identified in some breeds, but they are breed-specific; that is, the assortment of genes is different in every breed. (For example, see studies on the German Shepherd dog (Marschall & Distl 2007, Fells & Distl 2014, and Fels et al 2014), Bernese Mountain Dog (Pfahler & Distl 2012), and Labrador Retriever (Phavaphutanon et al 2008). Genes that could cause hip dysplasia have not been found in any breed.


It's unlikely that researchers are going to discover an easy genetic solution to the problem of hip dysplasia. It is a complex trait that is influenced by both genes and environment, and there is no simple solution just over the horizon. We should be able to improve genetic progress by using selection strategies that are as efficient and effective as possible such as estimated breeding values, EBVs. One great advantage of using EBVs is that the genes responsible for a trait don't need to be known; you need only a pedigree database and information about affected animals.

3) Environmental factors are also important

Although there is a genetic influence on hip dysplasia, the heritability of the trait is rather low. Many studies have shown that genetic variation accounts for only a modest fraction of the variation in hip scores, usually 15-40%. This means that some fraction of the variation in the quality of the hips is the result of non-genetic, or "environmental" influences. This is one reason why decades of strong selection has resulted in only modest reductions in hip dysplasia in some breeds. At the current rate of progress and selecting only by phenotype, it could take decades to achieve a meaningful reduction in the incidence of hip dysplasia (Lewis et al 2013).


Understanding the specific environmental factors that play a role in the development of hip dysplasia should allow us to reduce the number of animals affected by hip dysplasia even if the genetic basis is not yet understood. This would reduce significant pain and suffering as well as the expense and heartache endured by owners of an afflicted dog. There is no reason why we should not be taking active steps to do this now.


​The top three environmental factors that have been found to play a significant role in the develop of dysplastic hips are: a) joint laxity, b) weight, and c) exercise (see below).

4) Joint laxity is the primary cause of hip dysplasia

Puppies are born with perfect hips, and if the hips do not develop laxity the dog does not develop hip dysplasia (Riser 1985). Joint laxity occurs when the head of the femur does not fit snugly into the acetabulum. This could be the result of traumatic injury, overloading of the joint by weight, lack of muscle strength, or adductor forces (e.g., bringing the legs together). Joint laxity is the primary factor that predisposes a dog to the development of hip dysplasia.


​In dogs as well as many other vertebrates (including humans), the head of the femur in newborns is held securely in place by a strong ligament variously called the "round ligament" or "teres ligament".


One end of this ligament is attached to the head of the femur and the other end to the inner wall of the acetabulum (the cup-like socket on the pelvis).


You can see the teres ligament in this illustration (labeled "LIGAM. TERES"). 



If this ligament is damaged or severed, the femur will not be held tightly in the socket, which will cause the joint to feel "loose".



If the femoral head is not positioned properly in the socket, the forces on the hip will be abnormal. Instead of being distributed across the inner surface of the socket, the forces on the joint will be concentrated in a smaller area on the weaker rim of the acetabulum. The result will be damage to the rim of the socket when a load is placed on the hip joint.




5) Controlling joint stability is key

​The teres ligament should hold the head of the femur securely in the socket of the growing puppy while the muscles that will support the hip develop and grow stronger. But in some puppies, the ligament shows evidence of damage before they are even a month old (Riser 1985).


"The teres ligaments of the hip joints were edematous [swollen], a few ligament fibers were torn, and capillary hemorrhage dotted the surface of the ligaments at the point of the tears. These changes were considered the first findings that might be linked to hip dysplasia."


​The abnormal forces on the femur and acetabulum that are caused by joint laxity result in the trauma that causes hip dysplasia and osteoarthritis of the hip.


"There is no evidence that a primary defect of bone exists but rather the disease is a failure of the muscles and other soft tissues to hold the hip joint in full congruity. This is further supported by the fact that bony dysplasia can be increased, decreased, or prevented by controlling the degree of joint instability and incongruity. No other malformations are associated with the disease. A causal relationship between muscles and soft tissue defects or pathologic changes other than lack of muscle mass or strength has not been established... Hip dysplasia is a concentration of factors from a pool of genetic weaknesses and environmental stresses that fall into a programmed pattern of progressive remodeling and degenerative joint disease." (Riser 1985)


6) Body weight is a MAJOR environmental factor

If there is laxity in the hip joint, the amount of damage done to the femur and acetabulum will depend on the magnitude of the forces in the hip joint. The heavier the dog, the greater the forces will be and also therefore the higher the risk of hip dysplasia and osteoarthritis.


​Puppies that weigh more at birth as well as those with higher growth rates (so they get heavier sooner) have a higher risk of degenerative changes in the hip joint (Vanden Berg-Foels et al 2006).


​As this graph shows, puppies kept on a restricted diet (gray line) have a dramatically lower risk of dysplasia and it develops much later in life than in puppies kept on normal rations (black line) (Smith et al 2006). 


At four years old, less than 10% of dogs kept on a restricted diet (25% less than the control diet) were dysplastic, while at the same time more than 30% of the dogs in the control group were dysplastic. As an added advantage, dogs on restricted diets live longer, too (Kealy et al 2002)!

Unfortunately, many dogs (including show dogs!) are overweight (McGreevy et al 2005, Corbee 2013), and obesity could well be the single most significant environmental factor affecting the development of hip dysplasia and osteoarthritis. But body weight is a factor that we can control.


​Although progress from genetic selection will take many generations, the incidence of hip dysplasia in dogs could be immediately and dramatically reduced simply by practicing better weight management.


7) Exercise is good and bad

Exercise strengthens the muscles of the legs and pelvis, and this will increase the stability of the hip joint. But all exercise is not created equal.


Puppies raised on slippery surfaces or with access to stairs when they are less than 3 months old have a higher risk of hip dysplasia,while those who are allowed off-lead exercise on soft, uneven ground (such as in a park) have a lower risk (Krontveit et al 2012). Dogs born in summer have a lower risk of hip dysplasia, presumably because they have more opportunity for exercise outdoors (Ktontveit et al 2012). On the other hand, dogs from 12-24 months old that regularly chase a ball or stick thrown by the owner have an higher risk of developing dysplastic hips (Sallander et al 2006). 


The most critical period for proper growth and development of the hip in dogs is from birth to 8 weeks old, so the type of exercise the puppies are exposed to is most important during this time.


8) Nutrition is important

While puppies are growing rapidly, it is critically important to get their nutrition right.


Growing puppies need to eat enough to support growth but they should not be fat, because any extra weight can increase the risk of developing hip dysplasia (Hedhammar et al 1975, Kasstrom 1975). An additional problem is that puppies getting too much food could also consume too much of specific nutrients. Puppies provided a quality commercial puppy food that is fed in the proper amount will have a nutritionally balanced diet and should not receive any supplements. Dietary supplements, especially of calcium, are not only unnecessary but could cause serious problems. There is no evidence that supplemental protein or vitamins will reduce the risk of hip dysplasia (Kealy et al 1991, Nap et al 1991, Richardson & Zentek 1998).


9) Early intervention is critical

Most treatments for hip dysplasia are easier and more successful in younger dogs. If early symptoms are overlooked and screening is done only after 24 months or more, the window of time with the best prognosis in response to treatment will have passed (Morgan et al 2000). Signs of lameness usually first appear when the puppy is 4 to 6 months old, but after a month or two the dog will often seem better. This is because damage to the acetabular rim such as microfractures will have healed and the dog is no longer in pain, but development of dysplasia and osteoarthritis will continue. From there, the dog might not display clinical signs again for years while the pathological damage progresses.


Laxity in the joint can be determined as early as 4 months old (either by palpation or PennHIP). If detected early, intervention to mitigate additional damage can include weight loss, modifying exercise and activities, or surgery - but it must be done early before skeletal growth is complete. Breeders should educate new puppy owners about the factors that can increase the risk of developing hip dysplasia and also advise them to get a veterinary examination immediately if there is any sign of lameness.


10) We can dramatically reduce hip dysplasia now

Genetic selection should continue to produce modest progress in the reduction of hip dysplasia. But a significant and immediate reduction in the number of afflicted animals could be achieved by better control of non-genetic, environmental factors. Weight management, appropriate exercise, proper nutrition, and early intervention at the first sign of lameness are simple steps we can take that will dramatically reduce the pain and suffering caused by hip dysplasia. The research will surely continue, but we already have the information we need to tackle this problem. 

Need to brush up on your hip anatomy? Check out the 3D virtual tour!

Virtual tours of the canine hip and pelvis


You can learn more about hip dysplasia in ICB's 10 week course

"Understanding Hip and Elbow Dysplasia".

The next class starts 4 January 2016.

Corbee RJ. 2012. Obesity in show dogs. Anim. Physiol. Anim. Nut. DOI: 10.1111/j.1439-0396.2012.01336.x


Fels L, Y Marschall, U Philipp, & O Distl. 2014. Multiple loci associated with canine h.ip dysplasia (CHD) in German shepherd dogs. Mamm. Genome. DOI 10.1007/s00335-014-9507-1.


Hedhammar A, F-M Wu, L Krook, HF Schryver, A de Lahunta, FP Whalen, FA Kallfelz, EA Nunez, HF Hintz, BE Sheffy, & GD Ryan. 1975. Overnutrition and skeletal disease: An Experimental Study in Growing Great Dane Dogs. Cornell Vet 64: Supp 5.


Kasstrom H. 1975. Nutrition, weight gain and development of hip dysplasia. An experimental investigation in growing dogs with special reference to the effect of feeding intensity. Acta Radiol Suppl 344: 135-179.


Kealy RD, DF Lawler, & KL Monti. 1991. Some observations on the dietary Vitamin D requirement of weanling pups. J Nutr  121: S66-S69.


Kealy RD, DF Lawler, JM Ballam, SL Mantz, DN Biery, EH Greeley, G Lust, M Segre, GK Smith, & HD Stowe. 2002. Effects of diet restriction on life span and age-related changes in dogs. J. Am. Vet. Med. Assoc. 220: 1315-1320.


Krontveit RI, A Nodtvedt, BK Saevik, E Ropstad, & C Trangerud.  2012. Housing- and exercise-related risk factors associated with the development of hip dysplasia as determined by radiographic evaluation in a prospective cohort of Newfoundlands, Labrador Retrievers, Leonbergers, and Irish Wolfhounds in Norway. Am J Vet Res 73: 838-846.


Lewis, TW, SC Blott, & JA Woolliams. 2013. Comparative analyses of genetic trends and prospects for selection against hip and elbow dysplasia in 15 UK dog breeds. BMC Genetics 14:16. 


Marschall Y & O Distl. 2007. Mapping quantitative trait loci for canine hip dysplasia in German Shepherd dogs. Mamm. Genpome 18: 861-870.


McGreevy, P D, PC Thomson, C Pride, A Fawcett, T Grassi, B Jones. 2005. Prevalence of obesity in dogs examined by Australian veterinary practices and the risk factors involved. Vet Rec 156, 695-702.


Morgan JP, A Wind, & AP Davidson. 2000. Hereditary bone and joint diseases in the dog. Schlutersche GmbH & Co. KG, Germany.


Nap RC, HAW Hazewinkel, G Voorhout, WE van den Brom, SA Goedegebuure, & AT Kloosteer. 1991. Growth and skeletal development in Great Dane pups fed different levels of protein intake. J Nutr 121: S107-S113.


Pfahler S & O Distl. 2012. Identification of Quantitative Trait Loci (QTL) for Canine Hip Dysplasia and Canine Elbow Dysplasia in Bernese Mountain Dogs. PLoS ONE 7(11): e49782. doi:10.1371/journal.pone.0049782


Phavaphutanon J, R.G. Mateescu, K.L. Tsai, P.A. Schweitzer, E.E. Corey, M.A. Vernier-Singer, A.J. Williams,N.L. Dykes, K.E. Murphy, GLust, R.J. Todhunter. Evaluation of quantitative trait loci for hip dysplasia in Labrador Retrievers. Am. J. Vet. Res. 70: 1094-101.


Richardson DC & J Zentek. 1998. Nutrition and osteochrondrosis. Vet Clinics of N Am: Small Anim Pract 28: 115-135.


Riser WH. 1985. Hip dysplasia. Ch 83 In Textbook of Small Animal Orthopedics. CD Newton & DM Nunamker, eds. 


Sallander MH, A Hedhammar, & MEH Trogen. 2006. Diet, Exercise, and Weight as Risk Factors in Hip Dysplasia and Elbow Arthrosis in Labrador Retrievers. J. Nutr. 136: 2050S-2052S.


Smith, GK, ER Paster, MY Powers, DF Lawler, DN Biery, FS Shofer, PJ McKellvie & RD Kealy.  2006. Lifelong diet restriction and radiographic evidence of osteoarthritis of the hip joint in dogs. J. Am. Vet. Med. Assoc. 5: 690-693.


Vanden Berg-Foels WS, RJ Todhunter, SJ Schwager, & AP Reeves. 2006. Effect of early postnatal body weight on femoral head ossification onset and hip osteoarthritis in a canine model of developmental dysplasia of the hip. Ped. Res. 60: 549-554.


* Revised 13 Dec 2015

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